Chapter 8 Superficial and deep perivascular inflammatory dermatoses Chronic superficial dermatitis 259 Toxic erythema 261 Erythema annulare centrifugum 261 Erythema gyratum repens 263 Lymphocytic infiltrate of the skin 264 Reticular erythematous mucinosis 265 Polymorphous light eruption 267 Tumid lupus erythematosus 269 Perniosis 270 Chilblain lupus erythematosus 272 Pigmented purpuric

One hundred ninety patients with a clinical or histological diagnosis of urticarial dermatitis or both were stratified into 3 groups: group A, in which 49 patients (25.8%) had a clinical and biopsy-proved diagnosis that matched; group B, in which 99 patients (52.1%) had a clinical diagnosis that was other than urticarial dermatitis but in whom the biopsy result showed urticarial dermatitis

Allergic contact dermatitis occasionally provokes atypical T-cell infiltrates which may simulate mycosis fungoides. Contact eczema pathology Five major pathological patterns were identified: lichenoid (45/107, 42.1%), perivascular (40/107, 37.4%), interface (11/107, 10.3%), spongiotic (7/107, 6.5%) and granulomatous (4/107, 3.7%). Lymphocytic vasculitis was present in 17 patients (15.9%), and Langerhans cell microabscess was seen in 4 (3.7%). Perivascular lymphocytic infiltrate. Perivascular lymphocytic infiltrate refers to inflammation centred around the small vessels in the skin.

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The appearance of many skin diseases vary at different stages of their development and may be altered by attempted treatment and secondary changes such as scratching or infection. PERIVASCULAR INFILTRATES See DX Path for details of the conditions below Sometimes when you look at a slide the first thing you notice is that there is an infiltrate around vessels which can be superficial or deep or a combination of both. A pattern of subacute, chronic dermatitis or acute dermatitis may be seen. The dermal inflammatory infiltrate predominately contains lymphocytes and other mononuclear cells (figure 7). Allergic contact dermatitis occasionally provokes atypical T-cell infiltrates which may simulate mycosis fungoides.

deep, and cellularity b. Types [see slide 25]: i. Spongiotic = eczema ii.

Jan 18, 2021 Skin nontumor - Acute eczematous dermatitis. Spongiosis, perivascular lymphocytic infiltrate; Superficial dermal edema. Differential 

There isn’t much difference between perivascular dermatitis and other types of dermatitis really; it’s just that this one occurs near the blood vessels, caused by irritation to the skin or allergens. Perioral dermatitis is a relatively common inflammatory disorder of facial skin, often appearing in patients with rosacea, but with less inflammation. A typical perioral dermatitis presentation occurs with the eruption of papules and pustules confined to the nasolabial folds and the skin of the chin … Differential diagnosis of interstitial granulomatous dermatitis. Leucocytoclastic vasculitis: Neutrophils and dust in interstitial granulomatous dermatitis are more widely distributed throughout the dermis in addition to around vessels.

JPC SYSTEMIC PATHOLOGY. INTEGUMENTARY SYSTEM. SEPTEMBER 2019. I-M06 . Signalment (JPC # 21474-29): Dog, breed not specified. HISTORY: This dog had bilaterally symmetrical, rugose thickening, lichenification, and gray-black hyperpigmentation of the axillary skin.

Intradermal mucin. Thin epidermis. Pathological changes may arise in epidermis, dermis and/or subcutaneous tissue. The pattern of changes may allow a diagnosis to be made or it may be non-specific. The appearance of many skin diseases vary at different stages of their development and may be altered by attempted treatment and secondary changes such as scratching or infection.

Both also show vacuolar degeneration of the basal layer. In contrast, the chronic lichenified AP lesions are associated with marked hyperkeratosis, acanthosis, elongation of the rete ridges, and tissue repair. PREDOMINANTLY PERIVASCULAR DERMATITIS Superficial perivascular dermatitis Pigmented purpuric dermatoses Pigmented purpuric dermatoses (PPDs) comprise asymptomatic to mildly pruritic pigmented lesions, with distinct clinical and histological findings.6 Although the aetiopathogenesis is unknown, ster-oids, antihistaminics, griseofulvin The histopathologic findings from the active border may show a vacuolar degeneration of the basal cell layer, occasional colloidal bodies, pigment incontinence, phagocytic cells, congestive and dilated vessels, and a perivascular inflammatory lymphohistiocytic infiltrate. 1-4 Fibrosis has not been reported as a histological feature for the diagnosis of ashy dermatosis.
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Microscopic.

Low power view of leukocytoclastic vasculitis gives the pattern of a busy dermis with a superficial and mid perivascular inflammatory pattern (Figure 1). In non-bullous cases, perivascular and interstitial neutrophils are sometimes present in the upper dermis, with damage to blood vessels An interface dermatitis with vacuolar alteration, not otherwise specified, may be caused by viral exanthems, phototoxic dermatitis, acute radiation dermatitis, erythema dyschromicum perstans, lupus erythematosus and dermatomyositis. Histology of lichenoid drug eruption, as well as LP, reveals a nonspecific lichenoid interface dermatitis, basal keratinocyte apoptosis, and pigmentary incontinence. 14 Microscopic findings that are more typical of a lichenoid drug eruption include the presence of eosinophils and plasma cells, a deeper perivascular infiltrate, and a higher proportion of necrotic keratinocytes than seen in JPC SYSTEMIC PATHOLOGY.
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skin pathology pathology in outline format with mouse over histology previews.

Exocytosis of the inflammatory cells is present, and there is no acanthosis or parakeratosis. In allergic/contact dermatitis and atopic dermatitis, eosinophils may be present in the dermis and epidermis (eosinophilic spongiosis). Polymorphic light eruption (PMLE) is a form of photosensitivity, which usually occurs in younger females.It is more common in patients who receive only intermittent sun exposure and typically consists of crops of papules, vesicles or plaques. Predominantly, a superficial perivascular lymphocytic infiltrate, extravasation of erythrocytes, and focal interface changes characterized DRESS cases.